Max in WT synaptoneurosomes, suggesting that Src signaling might be downregulated in KI synapses. Conversely, our power to rescue SERT function in KI midbrain synaptoneurosomes via the inhibition of FAK implies elevated FAK signaling downstream on the Pro32Pro33 mutant, as confirmed by enhanced pFAK localization in five-HT synapses. Our info https://erici543zpe1.blogsmine.com/profile